Macroautophagy in Parkinson’s GPR37: Critical Analysis of Scientific Process

Brief Summary

The research paper deals with specific pathophysiologic pathways to better explain Parkinson’s Disease, wherein overexpressed (GPR37) protein bodies are observed in abundance. In detail, it takes into account the probability that accumulation of unfolded protein aggregates is not sole definitive cause for such degenerative status. Through a combination of laboratory techniques and embryonic kidney samples, outcomes of the study reveal that human body’s compensating mechanisms increase significant protein degeneration in motor neural functions. Additionally, several conjecture are presented, such as whether similar protective processes are adequate compensations or other controlled conditions can induce excess GPR37 protein or not.
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